|
|
本帖最后由 l.w._007 于 2011-8-9 21:40 编辑
此子和他的阳性伴侣，尝试过一系列活动。以下是问答部分，问：我和一HIV阳性发生了关系，我知道，唾液不传播艾滋，嘴巴上有一小伤口，如果我吞下了HIV病毒，会传染吗？答：不会。
问：我是gay，有天晚上，另一个男的射了以后，我用他的精液来给自己打飞机，你知道的，这不是个明智的宪法，我的阴茎当时是发炎的，第二天早上，他射了，我又这么着了，这次，阴茎发炎的更厉害了。那天稍微晚点的时候，发现JJ上有红点。第二天，JJ上伤痕。假设，我撸的时候太用力了，让龟头破皮了，而且那精液沾上去了，这样的行为能传染HIV吗？答：HIV不能在宿主体外保持繁殖和感染的能力，因此不能。HIV一旦离开人体，温度，空气，PH值就会破坏HIV外壳，blablabla。
问：最近，接受了带套肛交，他射了之后，我坐在他阴茎上（没有套子），自己打飞机，他的阴茎没有插进我的身体里，但是就抵在我肛门上，肛门上可能因为刚才的肛交留下了伤口。后来他告诉我，他是阳性。虽然，我帮他他WTKJ了，这个感染几率很低。我想问，有没有坐在他YJ上感染HIV的可能。我有伤口的肛门和他的YJ接触了。我来问，就是看有没有必要吃阻断药，不想吃药。我知道，这不可能传染。答：你没有进行无保护的插入式性jiao，没风险。
问：我躺在阳性伴侣的身上，我身上有几小时前的伤口，可能就是表皮破了，但显然，皮肤不完整。之后阳性伴侣用他的YJ，在我的伤口上磨蹭。我想知道，他的前列腺液会不会通过这些伤口感染我。
答：你没有进行无保护的肛交，就不会，blablaba。不需要检测，没风险。
问：我和阳性伴侣进行了有套肛交，无套口交，我知道口交风险很低，低到不用去考虑，但是，我口里边有炎症，有破损，开始口交的时候，肯定就有了。我昨天把舌头也咬伤了。因为口交风险为万分之一，有伤口，炎症的就跟口腔溃疡是得，这种情况会让这种低位行为变得高危吗？答：不会，blablabla，HIV不通过口交传播。
此子最后为仍为阴性。。你们恐的东西。。没必要了吧？
还有一人，和阳性口交之后，发的帖子。明天翻出来，先贴出来。标题是，用来安抚你们对于口交的恐惧--为什么Teak和DR.HHH/HOOK是对的的原因。等不及可以先翻，这个记录的MERCK关于KJ的研究。包括口腔的结构，细胞，免疫功能，等等。
那些CDC的感染力子，实际上进行了比KJ更危险的其他行为，包括无套肛交和性jiao。
Anxiety reliever for all of you Oral worry-wells - Why Teak and DR. HHH/Hook are right! by fourfiftyone, Jun 10, 2008 10:07PM
After starting to worry irrationally again today I did a little more digging, and found an AMAZING ppt. presentation discussing and stating pretty much all of the studies on oral sex HIV transmission risks.
I never fully understood why TEAK, Dr. HHH and Hook , and the others that told me that my one time oral sex exposure with NO ejaculation was no/zero risk. I thought that if my partner has HIV, that if I had little cuts I didn't know about that I could have gotten HIV. While it is theoretically possible (because NOTHING is 100%... freak accidents happen), the ppt. presentation gave great info as to why it probably doesn't happen (at least under normal circumstances).
They state that a total of potentially 38 cases have been stated as oral (rec) led to infection. But they state it is likely that many (or all) of them probably had other risk factors that were more risky. The state that the CDC claims that there also have been a few cases of infection w/o ejaculation as well, but once again, these are probably unreliable or almost definitely attributable to other MAJOR risk factors such as suppressed immune depression such as during chemotherapy, major oral cuts, meth mouth, other std inflamm/infection, and probably not due to tiny cut/sores (think about it almost everyone has little cuts yet we see barely any or NO cases of infection w/o ejaculation).
Indeed, sites such as the online MERCK manual (very reliable) state that rec oral sex w/o ejac is only a theoretical risk and there had been NO documented cases.
The ppt. presentation offers the following info as to WHY oral transmission is so rare or pot. doesn't even happen (at least under typical conditions):
"Oral tissues are naturally resistant to HIV infection, unlike anorectal and genital tissues.
Mechanisms involved are not completely understood.
The innate immune response is a key defense against HIV-1 particularly at mucosal surfaces.
Mucosal epithelium provides a physical barrier to infection and also produces anti-HIV-1 peptides and proteins that serves as key effector molecules.
Jana NK, Gray LR, Shugars DC HIV-1 Stimulates the Expression and Production of Secretory Leukocyte Protease Inhibitor (SLPI) in Oral Epithelial Cells: a Role for SLPI in Innate Mucosal Immunity. J Virology; 2005:6432-6440
Oral antimicrobial factors having anti-HIV activity include secretory leukocyte protease inhibitor (SLPI), human beta defensins (hBDs), salivary agglutinin, thrombospondin 1, and mucins.
SLPI participates in mucosal defense by reducing inflammation, blocking the growth of selected bacteria, fungi, and non-HIV-1 viruses, HIV-1 and enhancing wound healing.
Jana NK, Gray LR, Shugars DC HIV-1 Stimulates the Expression and Production of Secretory Leukocyte Protease Inhibitor (SLPI) in Oral Epithelial Cells: a Role for SLPI in Innate Mucosal Immunity. J Virology; 2005:6432-6440
Brief exposure of cells to HIV-1 leads to a significant increase in SLPI mRNA and protein secretion, which occurs rapidly after contact with the virus.
The time frame, as little as 5 minutes, has biological relevance in that it mirrors the brief duration that oral epithelial cells are likely to be exposed to virus in vivo during receptive oral sex.
Given the anti-inflammatory and antiviral properties of SLPI, the induction of SLPI in virus-stimulated cells represents a tug-of-war between the virus and the host immune response, as the virus attempts to stimulate the local inflammatory response while the inhibitor tries to dampen the response and/or protect neighboring cells against infection.
An imbalance between the opposing responses may dictate whether virus exposure ultimately results in productive infection or protection.
Jana NK, Gray LR, Shugars DC HIV-1 Stimulates the Expression and Production of Secretory Leukocyte Protease Inhibitor (SLPI) in Oral Epithelial Cells: a Role for SLPI in Innate Mucosal Immunity. J Virology; 2005:6432-6440
"
So think about it this way:
With no ejaculation, HIV is present in a relatively small amount and may or may not even be infectious (or enough there to be infectious, although it likely is)). The low/small amount of HIV would then have to get past ALL of the above defenses and then you would need a direct route to the blood stream ON TOP of those defenses, and then EVEN THEN it would be likely that transmission wouldn't occur!
I found this read very interesting and it is GREAT support that HIV infection in receptive oral sex (especially without ejaculation) is either so extremely rare, or doesn't occur at all (IMHO I think with ejaculation probably does occur very,very,very rarely, and that oral sex w/o ejaculation either doesn't occur, or only has uber rarely under extreme extenuating circumstances).
Comments and opinions are greatly appreciated!
|
|
[复制链接]
IP卡
狗仔卡
发表于 2011-8-9 21:24
提升卡
置顶卡
沉默卡
喧嚣卡
变色卡
显身卡
楼主
就让那往事随风！肯定阴！
